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Special Issue Title:

Exosomes and Delayed Encephalopathy After Acute Carbon Monoxide Poisoning (DEACMP)

Deadline for manuscript submissions: 15 September 2021

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Special Issue Editor

  • Guest Editor

    Dr. Yuan JiangE-MailWebsite

    Attending doctor, Lecturer, Department of Rehabilitation Medicine, Clinical Medical College and The First Affiliated Hospital of Chengdu Medical College, Chengdu, China

    Interests: Neurological rehabilitation, Cardiovascular rehabilitation, Hyperbaric oxygen medicine

Special Issue Information

Acute CO poisoning is the most common type of acute poisoning worldwide, and about 40 % of surviving patients develop delayed encephalopathy after acute carbon monoxide poisoning (DEACMP). The symptoms of DEACMP include impaired judgment, poor concentration and memory, cognitive dissonance, personality changes, psychosis, and even Parkinsonism symptoms. To date, the mechanisms of DEACMP remain unclear.

Exosomes are a heterogeneous group of nano-sized natural membrane vesicle released from various cells and exist in body fluids. A growing amount of evidence shows that exosomes involve in pathogenesis, diagnosis and treatment modalities of some brain disease, such as ischemic stroke, Alzheimer's disease, Parkinson disease, multiple sclerosis, brain cancers, and so on. In the light of exosomes can overcome the blood-brain barrier (BBB), exosomes as therapeutic drug carrier for brain drug delivery, have received extensive attention.

Previous studies suggested that DEACMP may be the outcome of acute CO poisoning induced cerebral cellular hypoxia, postischemic reperfusion injury, free radical damage, immune damage, and apoptosis. The secretion of exosomes by cerebral cells is mediated by various physical, chemical, and biological stimuli such as hypoxia, low pH, extracellular ATP, increased intracellular Ca2+, and DNA damage. Therefore, the potential roles of exosomes in pathogenesis and prognosis of DEACMP is worth deep exploration.

In this issue we welcome original research articles and reviews on this topic to provide recent findings and explore diverse functions of exosomes in DEACMP.

Keywords

Acute CO poisoning, DEACMP, Exosomes

Manuscript Submission Information

Manuscripts should be submitted online by submit system. Manuscripts can be submitted until the deadline. All papers will be peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Original articles, case reports or comprehensive reviews are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website. Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a double-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Signa Viate is an international peer-reviewed open access semi-annual journal published by MRE Press. Please visit the Instructions for Authors page before submitting a manuscript.The Article Processing Charge (APC) for publication in this open access journal is $1200. We normally offer a discount greater than 30% to all contributors invited by the Editor-in-Chief, Guest Editor (GE) and Editorial board member. Submitted papers should be well formatted and use good English.

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